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The CO stretch signal was observed in the DRIFTS as the Cu2O phase appeared in the simultaneous XRD pattern.
For example, contracting a muscle fibre elicits a proprioceptive stretch signal in a one-to-one fashion.
SACs and microtubules appear to play a role in transduction of the stretch signal to more downstream located pathways, which appear to be similar in PPC and other modes of postconditioning.
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This means the stretched signal will have multiple folds.
A strong vibrational stretching signal at 1270.1 correspondsponds to the urethane-like amide bond of the linker [41].
Furthermore, a strong broad signal between 3,000 and 3,650 cm−1 is present, attributed to the stretching signal of the SiO-H bond [22].
It could be related to the stretching signal of Si-O-H bonds regardless of the Q n specie carrying those bonds.
However, recent results from molecular dynamic simulations (Spiekermann et al. 2012) questioned the attribution of the 976 cm−1 signal in pure hydrous silica and suggested that this peak may actually arise from the asymmetric Si-O stretching vibration of Q 2 units and that the Si-O-H stretching signal may form a shoulder near 920 cm−1 on the side of the 976 cm−1 peak.
Higher temperatures drive ligand desorption until 200 °C where 90% of the C H stretching signal from CdSe-CdCl 2/PBu 3 disappears.
At 323 K, we clearly observe a decrease in intensity of the CH2 stretching signal, whereas the CH2 deformation at 1450 cm 1 and the CH vibration at 2791 cm 1 completely vanish.
As is shown in Fig. 8, the characteristic asymmetric stretching signals of Si O Si Bridge distribute between 1,000 and 1,300 cm−1 in the spectrum.
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