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An ompA-negative E. coli strain invaded brain microvascular endothelial cells only with difficulty [37], [38].
This meant that either the Wolbachia strain diversity was maintained in almost all repetitions or that one strain invaded both insect populations.
In this scenario, an ancestral Wolbachia strain invaded the first filarid host, evolved a mutualistic association that included strict vertical inheritance, and the C and D supergroups originated through ancient host lineage divergence.
Similar(56)
As the new strain invades the population it leaves a molecular signature in genetic sequences sampled randomly from the pathogen population.
Some strains invaded the host cells as soon as 15 minutes, and the invasion increased according with time.
Furthermore, these naturally occurring SPI-1 deficient strains invaded human monolayer cells less efficiently than laboratory constructed SPI-1 mutants, further demonstrating differences between monolayer systems and in vivo outcomes of infection.
Grant et al. [ 26, 27] showed biotype 1A strains invaded epithelial cells and resisted the killing effect of macrophage.
In this case, probably several Rex3 strains invaded the genome of these species and have been retained, with gain and loss of sequences within each species.
Thus, we investigated whether mT3 E. coli strains invade nonphagocytic HeLa cells.
Current definitions are becoming of decreasing usefulness as typical CO-MRSA strains invade hospitals.
If so, the similarity is of particular interest with regard to potential infection powers in human and/or swine populations when these strains invade these populations.
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