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We have two rectangular slip surfaces, one for each stage of the strain event that consists of contraction followed by expansion.
The data do not preclude continuing slip over a longer time interval as has been shown by Ozawa et al. (2004) but since we cannot readily recognize such strain changes we limit our analysis to the clearly observable strain event.
The resulting signal decomposition provides a corrected strain signal, showing a clear strain event consisting of contraction for about 4 days, and then expansion for about 23 days, following the 2003 Tokachi-oki earthquake.
We assume, as did previous investigators, that the source for this strain event is coplanar with the mainshock and additionally that the slip is in the same direction as the coseismic slip and is uniform over our entire model surface.
A Sacks-Evertson borehole volumetric strainmeter (SE strainmeter) at a site located 105 km from the epicenter of the mainshock recorded a clear slow strain event following the 2003 M w 8.0 Tokachi-oki earthquake (September 25 , 2003 19 50 06 UTC).
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Specific strain events were attributed to the gelation and vitrification phases of the epoxy cure cycle.
Acute local strain events can involve a twofold extension of a single SF sarcomere, but how these dramatic local events affect the overall SF architecture is not believed to be understood.
Analysis of the recordings revealed that the measured bone surface was subjected to 2.9 (± 1.4) × 103 strain events per hour of which 1.8 (± 1.0) × 103 had amplitudes ≤ 10 microstrains.
We suggest that high sensitivity fluid flow meters can be utilized to detect transient tectonic strain events in offshore environments where traditional geodetic techniques lack resolution or are not possible.
This paper presents a new method for crack growth detection in steel plates through the analysis of cumulative duration of strain events measured by a self-powered sensing paradigm at preselected level discretization.
Studies of cellular response after blast and strain events provide evidence of degenerative processes in the neuron itself (Leung et al. 2008; Duncan et al. 2011).
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