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This study expands on existing modelling approaches by allowing for varying stock sizes within patches and stochastic spread between patches.
We present in this article a method to assess a system efficiency based on modelling of the temporal and stochastic spread of faults.
Can these conclusions really be drawn from the agreement between the node degree distribution, the split off nodes and the stochastic spread of the data when comparing the actual data for metabolic networks and the blind watchmaker model network?
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We specifically present a lower-bound on the decay rate of the number of nodes infected by a susceptible infected susceptible (SIS) stochastic spreading process.
PEV occurs when a gene located near a euchromatin-heterochromatin border is randomly silenced or expressed due to the stochastic spreading or contracting of heterochromatin.
Based on these earlier observations, together with the sensitivity of PEV to the dosage of chromatin modifiers, it has been thought that PEV results from the stochastic spreading of heterochromatin-forming factors or heterochromatic chromatin marks from a nearby heterochromatic region into the variegating gene, in the absence of dominant cis-acting boundary elements [ 37- 39].
Here we develop an objective way to fit simple stochastic fire spread models based on readily available data from documented fire events (i.e. approximate ignition point, preexisting vegetation, final perimeter, topography, and average wind direction).
We estimate this stochastic energy spread leads to almost no change in uniformity of temperature for the target heated by the Li beam, but leads to a factor of two of non-uniformity in temperature of the target for the K beam over a depth of 3.4μm.
In this paper, a neural model that accounts for stochastic firing, parasitic spread of excitation across neuron populations, and neuronal refractoriness, was developed and augmented as a preprocessing stage for a standard 22-channel noise-band vocoder.
Here we develop a stochastic model for vCJD spread via primary transmission through the consumption of BSE contaminated beef and secondary human-to-human transmission through red cell transfusions (but ignoring other blood components and transmissions via surgery or dentistry), taking into account the genetic structure of the population at codon 129 of the PrP protein.
This suggests that the differences observed between different countries are observed due to the stochastic nature of influenza spread.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com