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Each stimulating session lasted 2 min (50 stimuli every 2.4 sec = 120 sec = 2 min).
One conclusion that could thus be reached from observing this stimulating session is that genetic and epigenetic changes probably intersect at the microenvironmental level to facilitate architectural changes that ultimately result in the emergence of the malignant phenotype.
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A clear increase of heat pain threshold was observed in the skin territory of the left radial nerve when this nerve was stimulated (session 1).
Han Brunner (Radboud University, Netherlands) stimulated post-session discussions centered on attributing causality, phenotyping, and integration of genomic science into clinical medicine.
During stimulating and captivating sessions we explored the new frontiers in neuroscience.
The session stimulated students' interest in recognising abnormalities, working with simulated patients, revision and clinical skills/practice.
Left side and right side were stimulated 15 times per session in each patient (triple pulse, monopolar square wave, duration 0.5 ms, pulse interval 5 ms, interstimulus interval: 12 to 18 seconds, pseudo-randomized).
Left side and right side were stimulated 15 times per session in each subject before and after smoking/caffeine consumption (triple pulse, monopolar square wave, duration 0.5 ms, pulse interval 5 ms, interstimulus interval: 12 to 18 seconds, pseudo-randomized).
The extra active control condition was included later (in 10 subjects), this area was always stimulated in the last session.
Students received a written summary of the first session to stimulate elaboration and discussion in the second session.
A short presentation (5 10 minutes) on the topic was given at the beginning of each session to stimulate discussion.
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