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The proliferative mechanism for early relapse, which is also the result of surgery, is stimulated division of single dormant cells [ 8] or even changes in the dynamics maintaining the steady state of dormant or indolent micrometastases, eventually resulting in angiogenesis and growth.
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"We also reject [the idea] that anyone uses religion to stimulate division between us, Muslims and Christians," he added.
Applied electric fields have been shown to stimulate division, migration and differentiation of many types of cells: corneal epithelial cells [15] [18], neuronal cells [19] [21], fibroblasts [22] and skin epithelial cells [23] [24] in a process called galvanotaxis or electrotaxis.
We have suggested that one of the side effects of surgery is to stimulate division of dormant single malignant cells and stimulate angiogenesis of dormant micrometastases.
108, 109 The possibility of stimulating β cell replication has also been receiving considerable attention, with the identification of new compounds that can stimulate division 110, 111 and the elucidation of mechanisms that might be exploited in the future.
They hypothesize that there is induced angiogenesis after breast cancer surgery in approximately 20% of premenopausal node-positive patients, which stimulates division of dormant micrometastatic cells and synchronizes them into a highly chemosensitive state.
This increase in cell division most likely occurred due to the chemical composition of the fruit, particularly the relevant vitamins and sugars that stimulated cell division in normal cells from the bone marrow of treated rats.
We also determined whether FFO stimulated cell division.
With increasing dosage of radiation, stimulated cell division accompanied by increase in nuclei size but reduction in cytoplasm was observed.
The overall findings pointed to stimulated cell division and tissue repair mechanisms as the underlying cause of resistance.
The objective of the current study was to investigate if the antimitotic effect of colchicine (CLC) abolishes this resiliency to CD + CCl4 by inhibiting ongoing and stimulated cell division.
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