The results suggest that there are thermosensitive receptors in the faucial pillars that promote swallowing when stimulated by cold touch.
Genotypic variation was present among hybrid combinations in the production of unreduced gametes in Brassica interspecific hybrids, and some hybrid genotypes were stimulated by cold temperatures to produce high levels of unreduced gametes.
In Clade I, the expression of both Os04g45750 and Os02g43300 were stimulated by cold, salt, and drought conditions, whereas expression in the sorghum homologs, Sb06g023980 and Sb04g033390, were not influenced by any of the stressors.
While multiple tissues are simultaneously sympathetically stimulated during cold exposure, there are many examples of treatments that result in differential sympathetic outflow to various types of tissues (e.g. WAT vs BAT) and even within a type of tissue (e.g. different WAT pads) [ 24].
For example, within the list of genes responding to dehydration-stress, many genes have been also recognized as components of pathways stimulated by cold, temperature, light, and radiation (~750 genes), by hormones, endogenous, and chemical stimuli (~300 genes), as well as genes encoding transcription factors and regulators (131 genes).
Sb03g038280 was stimulated only by cold.
It showed an expression pattern similar to the BAT marker gene Cidea in that expression was greater in BAT vs. WAT and that cold stimulated a large increase in WAT, whereas only a small increase was detected in BAT.
Thus, we suggest that the AsA-GSH cycle is involved in mechano-stimulated cold tolerance in winter wheat.
The findings might provide deeper insights into roles of reactive oxygen species in mechano-stimulated cold tolerance of photosynthetic apparatus, and be helpful to explore novel approaches to mitigate the impacts of low temperature occurring at critical developmental stages.
In a piece of good economic news, chain store sales rose as cold weather stimulated demand for winter clothing, Instinet Research said in its weekly Redbook report.
In two genetic mouse knockout models (apolipoprotein E−/− [ApoE−/−] and LDL receptor−/− [Ldlr−/−] mice), persistent cold exposure stimulated atherosclerotic plaque growth by increasing lipid deposition.
