This article tries to stimulate further dynamic intermittency studies on the issues caused by renewable intermittency, which have not been revealed because of inadequate/incomprehensive study methodologies.
This review focuses on the factors that facilitate and stimulate the dynamic assembly of metabolons, including microenvironments, noncatalytic proteins, and allosteric regulation.
Among them, the interaction of cofilin and actin-depolymerizing factor (ADF) with monomeric and filamentous actin is of paramount importance, since they stimulate the dynamic behavior of actin filaments (Condeelis et al. 2001b).
The working model we arrive at (described below) helps to explain how >30 different actin regulatory proteins arrive at patches within 5 7 sec of each other and work in concert to initiate the rapid formation of an actin network, stimulate its dynamic turnover and remodeling, and ultimately dismantle the network and recycle its components.
HIV-1 stimulates dynamic and complex responses within macrophages through alteration of expression or modification of cellular genes or proteins.
The protein overexpression of actinin-4 in cancer cells stimulates dynamic remodelling of the actin cytoskeleton, and it is for this reason that actinin-4-overexpressing cancer cells have metastatic potential (Hayashida et al, 2005).
This article stimulates further dynamic intermittency studies on the issues caused by renewable intermittency.
Redifferentiation and proteoglycan synthesis could instead be stimulated by dynamic mechanical compression of the implant.
Cartilage explants from FrzB −/− and wild-type mice were stimulated by dynamic compression for 6 hours.
Hence, the goal has become less one of specific structural change and more one of stimulating a dynamic that enables the knowledge production process to become self-sustaining.
