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Each iteration of this second stage advances the system in time by a typically small timestep (Δ t=10−15 s), and thus complete simulations normally require up to 106−109 iterations.
The density of CRC inflammatory infiltrate declines as stage advances.
The second stage advances exchange by enabling medical information to be directly accessed by hospitals via the Health Data Bank, a dedicated server (Paths 5 and 6).
These observations suggest that as the tumor stage advances, the association between PIVKA-II and the risk for metastases may be weakened.
In summary, the density of overall CRC inflammatory cell infiltrate declines as tumour stage advances, likely depicting weakening immune response against the tumour.
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Finally, as expected, we found an association between advanced T stage, advanced node status and poor tumor differentiation.
The 5-year relative survival markedly decreased as the stage advanced.
Cortactin amplification was related to increased lymph node stage, advanced disease stage and reduced survival (Rodrigo et al, 2000).
We used the same three-step model described above, also including stage (advanced or early) as a variable.
Three patterns of up-regulation were found in trichome developing stage, advanced trichome elongation stage, and the trichome-cell-determining stage.
IHL-305 was administered on day 12 after the inoculation of ES-2 cells; at this stage, advanced tumor invasion (omentum, pancreas, etc).
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