The molecular mechanisms underlying the induction of adventitious roots to become SRs are, however, poorly understood.
Even after accumulation of large amount of experimental support, the exact genetic and physiological basis for SR bias is still far from understood.
How these diverse stretches are modified and what role they play in SR protein function is not well understood.
Although there exists research guidance on when and how to update SRs [ 6, 7], the process is not well understood.
Moreover, it is understood that more accurate results in estimation of SR are obtained in the use of satellite data, rather than the use of meteorological station data.
While the exact mechanism of delivery of cholesterol siRNA from HPPS is not fully understood, we speculate that this mechanism also involves SR-BI.
While SR Ca2+ release was extensively studied in MH [ 15], the impact of an altered SR Ca2+ reuptake on the pathogenesis of MH by intrinsic or extrinsic factors is poorly understood.
However, there are so-called 'steroid-resistant' (SR) patients who fail to respond to treatment with GCs [ 1- 3], the pathogenic mechanism of which is not fully understood.
An exact-match query q e is defined as q e = 〈 f ̂, κ ̂, ĉ, l ̂, E, Λ ̂ 〉, where f ̂ is a query required function, κ ̂ is the nonfunctional requirement, l ̂ is the legal rules requirements, ĉ is the contextual information of the service consumer, E is the authentication certificate and Λ ̂ is the set of parameters SR can provide or understand.
The contrary intuition underlying the SR model is that we understand both outcomes equally well.
We here use Sr-Nd isotopic compositions to understand the source of sediment in the river and the variations in the downstream influx of sediment both from local sources and from the larger tributary basins (Fig. 11).
