Sentence examples for spread virulence from inspiring English sources

Experimental crosses have now proved that all these pathogens have extant capacity for genetic exchange, with potential to generate new genotypes and spread virulence and drug resistance.

Because virulence-related genes could be transferred among bacteria by viruses (Beres et al. 2002), the ability to infect different spiroplasmas indicates that plectroviruses may help to spread virulence factors among different Citri species that share similar niches, such as the three leafhopper-transmitted phytopathogenic spiroplasmas (Liu et al. 1983; Whitcomb et al. 1986; Saillard et al. 1987).

Pathogenicity islands also play an essential role in spreading virulence genes through lateral gene transfer, an evolutionary process by which bacterial pathogens acquire new virulence factors en bloc.

Conjugative plasmids spread readily among Staphylococcus strains and to and from other genera such as Enterococcus (Lindsay 2010; Malachowa and Deleo 2010; Périchon and Courvalin 2009; Zhu et al. 2008), and they are implicated in spreading virulence loci and vancomycin resistance among clinical strains (Périchon and Courvalin 2009; Zhu et al. 2008).

While horizontal spread of virulence plasmids is not unusual, little is known about the lateral transfer of chromosomal B. cereus virulence factors [ 27, 29].

I was then studying the genetic regulation of Salmonella prophages and their role in the spread of virulence factors.

The authors, Dr. Susan Scott, a demographer, and Dr. Christopher J. Duncan, a zoologist, say their theory answers many lingering questions about the rapid spread and virulence of the Black Death.

The genetics and biochemistry of these proteins and others that affect endocytosis, targeting, virus assembly, egress, spread, and virulence are a major focus of our work.

ESX-1-related effects on host immunity include modulation of inflammation, apoptosis, necrosis, phagosomal maturation, phagosomal escape, membrane/cell lysis, tissue invasion, cell-to-cell spread and virulence.

These results suggest that NF-κB-mediated anti-apoptotic response is required for unrestricted EMCV replication, spread and virulence.

Antibiotic-mediated phage induction may result in high-frequency transfer and the unintended consequence of promoting the spread of virulence and/or antibiotic resistance determinants.