Sentence examples for spread of seizures from inspiring English sources

These operations, done mainly in the 1960s but recently reintroduced in a somewhat modified form, are a last-ditch effort to control certain kinds of severe epilepsy by stopping the spread of seizures from one lobe of the cerebral cortex to the other lobe.

This may facilitate further spread of seizures, since the thalamus has been shown to play an important role in seizure control [ 54– 54].

In epilepsy, α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptors play a predominant role in mediating the generation and spread of seizure activity whereas in migraine mainly N-methyl-D-aspartate (NMDA) receptors are involved in triggering CSD.

In addition, a reduction in seizure severity can result in a clinically meaningful success, if for instance spread of seizure activity e. g. generalization of focal onset seizures is prevented so that seizures remain focal.

Overexpression of AMPA receptors plays a crucial role in the forming and spreading of seizures.

In keeping with this hypothesis, Saji et al. (2000) reported that transient silencing of SuM hippocampal pathways, performed by injection of muscimol, an agonist of GABA-A receptor into the SuM prevents the genesis and spread of spontaneous seizures in a rat model of kainic acid-induced seizures.

5 7 Anomalous high-frequency repetitive spike firing, for instance, is known to take place during the spread of epileptic seizures, and there is an increasing body of evidence linking VGSC mutations with neuronal hyperexcitability in epilepsy.

Using these biomarkers, we reanalysed contemporaneous subdural EEG recordings of old surgical resection cases to show that this new approach does indeed provide a coherent interpretation of the spread of neocortical seizures (Weiss et al., 2015).

The whole 10 s window was used to model the spatial spread of the seizure.

Our aim here is to correctly model the spatial spread of the seizure using a 10-s EEG segment shown in Figure 5a.

Abolition of the hind limb tonic extension component of the seizure indicated protection against the spread of MES-induced seizures.