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As hepatic fibrosis progresses, the liver-to spleen density ratio may decrease because liver fat storage is depleted, but visceral fat is preserved.
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Using these values, we determined the liver-to-spleen density ratio.
The liver-spleen density ratio did not vary according to HCC etiology.
In contrast, no difference was noted in the liver-to-spleen density ratio according to HCC etiology.
Computed tomography images were used to measure the visceral fat area (VFA) and liver-spleen density ratio.
In contrast, the liver-to-spleen density ratio did not differ between the cryptogenic HCC patients and the HBV-HCC, HCV-HCC, and ALC-HCC patients.
VFA and the liver-spleen density ratio measurements support the theory that most cryptogenic HCC cases may be associated with burnt-out NAFLD.
The inverse of the liver-to-spleen density ratio was regarded as an indicator of the degree of fat infiltration in the liver [ 16].
Moreover, the liver-to-spleen density ratio tends to be lower in cirrhosis patients than in non-cirrhotic NASH patients [ 10].
As previously reported, the severity of fatty liver is positively correlated with VFA, whereas VFA and the liver-spleen density ratio are negatively correlated [ 9].
Based on VFA and the liver-to-spleen density ratio, cryptogenic HCC may be burnt-out NAFLD in which visceral fat remains but liver fat is depleted.
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