Sentence examples for spite of sequence from inspiring English sources

There were also examples of OLPs containing a BDE that were targeted in spite of sequence differences (i.e. OLP-139).

The T cell responses we detected to the CAPAGFAIL epitope were also of high avidity, so may have been better-able to cross-recognise our test peptide in spite of sequence differences to the autologous virus sequence than lower avidity responses to other viral epitopes.

Nevertheless, it should be noted that we could not clearly determine how many SNPs are doubtless due to RNA editing, sequence errors (in spite of sequence quality filtration), mapping error, or the reference sequencing errors.

In spite of sequence stacking due to low resolution of the linkage and physical maps, conserved syntenies can be identified although the exact gene order and orientation are unknown at present.

In spite of sequence homology, the IGFBPs may have quite different biological activity, due to differing abundances, with IGFBP-2, -4, and -5 predominating in the brain, and due to post-translational modifications [ 36, 37].

In spite of sequence differences near the glycan-binding sites, the mouse receptor closely mimics the properties of the human receptor, showing high affinity binding to glycans bearing terminal GlcNAcβ1-2Man motifs.

The incomplete sampling effort in spite of sequencing an average of 73,000 reads per sample (Figure S2), and the large number of distinct OTUs0.03 observed per quadrant (Table 1) clearly indicate a highly diverse microbial community in the spring, as previously suggested [20].

CNPs previously characterized by aCGH in D. melanogaster (Emerson et al. 2008) were also uncovered by 454 sequencing, in spite of low sequence coverage (∼0.2× per line) and the use of different fly strains.

Interestingly, in spite of eightfold sequence coverage of the mouse genome, no YKL39 sequence is available from mouse databases and there is no orthologous gene at the locus corresponding to the one where human YKL39 is located.

Finally, secondary structure predictions were performed on the aligned VDAC primary sequences, revealing a remarkable conservation in β-strand forming regions in spite of low sequence similarity in these segments.

Furthermore, the lead ligand exhibits selectivity against SH3 domains derived from other Src kinases, in spite of a sequence identity of approximately 80%.