Osteoporosis and osteomalacia are significant risk factors for fracture and spine instrumentation failure.
Previous attempts at bronchoscopic tracheal dilatation and stenting had failed because of a rigid cervical spine and failure to insert a rigid bronchoscope by cardiothoracic surgeons.
Systemic comorbidities included: hypertension; diabetes mellitus; joint and spine disorders; renal failure; cerebrovascular, cardiovascular, hepatic, pancreatic, or other organ disease; tumors/cancers; and histories of trauma and surgery.
This finding suggests that natural death of WT adult-born GCs might also be a result of the failure of spine formation in certain cell populations.
In the Discussion, the authors state: "this finding suggests that natural death of WT adult-born GCs might also be a result of the failure of spine formation in certain cell populations".
Hence, dendritic tau hyperphosphorylation and aggregation in sporadic Alzheimer's disease may be a secondary or even compensatory phenomenon due to slowly progressing deafferentation/disconnection in the aging brain rather than the cause of dendritic spine loss or synaptic failure.
The severe form of MPS VI, or rapidly progressive, is characterized by the onset of symptoms between 2 and 3 years of age, restricted joint mobility in infancy, delayed puberty, cervical spine compression, and respiratory failure.
These can be correlated with work on the injury patterns seen in blast to attempt to derive the posture of the spine at the moment of failure and to postulate the mechanism of failure [ 7, 12, 22].
This critical situation is responsible for a high risk of implant failures during spine surgery and of pseudarthrosis.
As Lambert et al. [9] mentioned in their study, scapular spine fracture represents a partial failure of the lateral scapular suspension system, leading to a failure of scapular postural control, with resulting sub-acromial impingement.
