This combination has the best stability value, but as normalization results show, it differs significantly from the artificial spike (p < 0.01, Table 6).
Given two spike trains made of M, N spikes, P = [ P 1, P 2, …, P M ] and Q = [ Q 1, Q 2, …, Q N ], and a time bin size B, for each time lag ∆T and for each spike P i (i = 1, 2,..., M) in P, we counted the number of spikes in Q that were within [ P i +∆T-B/2 P i +∆T + B/2].
Indeed, the actual somatic response would have been much more likely without the NMDA-spike, P ( Z [ t s, t s + Δ ] | Y ν ∖ { t ∗ } ) ≈ 0.72.
As expected, the shuffled spikes expressed no phase-preference, with k values significantly lower compared with recorded spikes (P < 0.001, Watson Williams test).
However, there was no difference in the instantaneous peak frequency between the two groups at the depolarizing current that evokes 30 40 spikes (p = 0.8221; Figure 7D).
Theta cells show a spike preference with the sniff phase, with k values for all recorded spikes significantly higher than the shuffled spikes (P < 0.001, Watson Williams test, Fig. 5D and H, left).
The values of circular coefficient k for the non-correlation group did not show statistical significance compared with the k values of the shuffled place cells' spikes (P = 0.28, Watson Williams test).
The distribution of the spike trains and bursts across the sniff cycle was phase-dependent (Fig. 5B and C), with higher k values (0.19 ± 0.02 for spike trains and 0.43 ± 0.01 for bursts, Fig. 5G middle and right, respectively) compared with shuffled spikes (P < 0.001, Watson Williams test).
Fig. 2 The spiked p-chain P8.
The association was higher for length of rachilla hairs, glume colour and spike density (P < 0.0001), followed by lemma colour (P < 0.001), row number (P = 0.003) and lemma type (P = 0.011).
The means averaged across groups exhibited a quadratic increase from session 1 to session 4: 91μV or 48% for mean spike amplitude (p <.05) and 7Hz or 16% for mean spike frequency (p <.05).
