It would be attributable to improper combination of the CGSSe absorber film and the CdS buffer layer in the conventional cell architecture, which may promote interface recombination due to an absence of a "spike" band alignment (i.e. conduction band offsets are slight positive values, 0.1 eV < ΔEc < 0.4 eV) with the CGSSe absorber layer.
This method is sensitive to both the existence and precise alignment of spike events in particular locations of the stimulus.
In addition, over the central region of the genome alignment, a spike centered on the receptor binding protein ∼17.5 kb and another region between 27.7 and 33.4 kb exhibit high recombination rates.
The spike gene amino acid alignment identified substitution at positions 23 (I), 31 (H), 57 (K), and 59 (E), which have not been identified in the complete PEDV genomes available from North America.
Specifically, sequence alignment indicated spike gene nucleotide deletions at positions 164 169 that correspond to amino acid deletions at positions 55 and 56 in addition to substitutions at positions 23 (I), 31 (H), 57 (K), and 59 (E) as compared to the CV777strain [ 10, 11].
To calculate the alignment between spikes and LFPs, we computed the LFP phase in theta band for each spike.
All other trains along the Zephyr's route were diverted to sidings and the turnouts were spiked into the proper alignment for the Zephyr's run.
The vertical alignment of each spike across trials indicates the high reproducibility of individual neuron spike times in a synfire chain.
This is evident from the rhythmic alignment of firing spikes of pyramidal cells in the spike raster plot (Figure 2D).
The alignment indicated a spike gene nucleotide deletion at positions 164 169 (TTGGTG), which corresponded to amino acid deletion at positions 55 and 56.
The top tables depict the total reads sequenced, and the alignment to the spike-in controls, ribosomal, reference genomes for each replicate.
