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A total of 20 specimens were randomized evenly into a stainless steel tension band group (control group); or a knotless tension band group.
32 ulnar nerve specimens were randomized into two groups: 18 nerves regenerating under the influence of recombinant growth hormone, and 14 nerves regenerating in its absence.
Specimens were randomized onto two plates with 80 specimens each, and laboratory personnel were blinded to case status at all times.
Specimens were randomized to receive various treatments (n = 6/group).
All the bone-disc-bone specimens were randomized into normal group, mimic group, and control group.
Specimens were randomized to receive the NO donor SNAP (100 μM), or the selective nNOS inhibitor TRIM (100 μM), for 20 min prior to ischemia in the absence and presence of the mitoKATP channel blocker 5-HD (1 mM) or the p38MAPK inhibitor SB203580 (10 μM) for 30 min. In addition, some preparations were subjected to IP.
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The following studies were sequentially carried out: To investigate this, specimens from ventricles of rat hearts were randomized to receive the exogenous NO donor SNAP (100 μM), the non-selective NOS inhibitor L-NAME (100 μM), or the selective nNOS inhibitor TRIM (100 μM), for 20 min prior to ischemia.
The studies were small and preliminary, and none were randomized, double-blind trials.
The groups were randomized.
Thirty-one patients were randomized.
Results Twenty-nine participants were randomized.
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