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In double-labeled specimens, we detected no apparent co-localization of GABABR2 immunolabeling and rdl-Gal4 expression in the distal portion of the lamina.
In the post-chemotherapy specimens we detected strong expression of GST-π in all but one of the samples.
Moreover, using EC tissue specimens we detected negative correlations between SHARP1 level and both lymphatic metastasis and markers of EMT.
In the relapsing and metastatic specimens, we detected the same alterations, as observed in biopsy or surgical samples, suggesting the evolution of the same tumour clone (Table 2).
Although we showed a co-infection rate of 71.4%, there were 8 (28.6%) of 28 patients with respiratory illness in whose specimens we detected only 1 virus, WUPyV.
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In one specimen, we detected 116 and 117 serotonin immunoreactive (5HT-ir) somata in the brain hemispheres, which are arranged in distinct cell groups.
When using different molecular targets and the dental pulp as a suitable specimen, we detected Y. pestis specific DNA fragments in European skeletons of persons suspected of having historical plague (3 – 5 ).
In the 205 specimens of NSCLC, we detected EGFR mutations in 66 (32%) samples, including exon 19 deletion mutations in 34 (17%) and exon 21 point mutations in 32 (15%) samples.
Even if only considering type specimens (thus possibly overestimating the date of first collection), we detected numerous specimens from South America dating back to the late 18th/early 19th centuries (following date intervals are given based on the documented time of explorations).
Of 50 patients who underwent bronchoscopy for workup of possible lower respiratory illness and had a BAL specimen available for testing, we detected KIPyV in 1 sample from a child who also had CMV pneumonia.
We detected >75 specimens of living and extinct bears affected with dental caries lesions.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com