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A heating ramp of 5 °C per minute was selected to expose the cylindrical specimens to target temperatures up to 800 °C.
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However, in the specimens exposed to target temperatures higher than 200 °C, the test results show that the epoxy coated rebar has less bond strength than the uncoated rebar.
At the age of 90 days, the specimens were heated to target temperatures of 100, 200, 300, 400, 500, 600, 700 and 800 °C respectively.
We used new cell line models to characterise novel chemotherapy resistance mechanisms and validated them in tumour specimens to identify new targets and biomarkers for gastroesophageal cancer.
Molecular diagnostic tests to detect enterovirus in clinical specimens usually target highly conserved sites in the 5′-non-translated region, allowing detection of all members of the genus.
Although we could not attempt isolation of microorganisms from the formalin-fixed, paraffin-embedded specimens, future studies targeted to mycobacteria would be especially useful in confirming our observations and in characterizing any association with sarcoidosis.
Three duplex real-time reverse transcriptase-PCR assays were run on each specimen to detect matrix gene targets specific for influenza A and influenza B, hemaaglutinin gene targets specific for influenza A subtypes H1 (seasonal), H1 (pandemic) and H3, and MS2 RNA coliphage (MS2) to monitor the efficiency of the assay [S. Williams, G. Chidlow, D.W. Smith, manuscript submitted].
In many ways resulting from the inability to selectively target bacteria in human specimens, ASTs typically rely on the lengthy process of monitoring growth inhibition of pure cultures under antibiotic challenge.
Since the rate of deposition of generated structures is proportional to the number of evaporated particles, denser structures are synthesized when specimens are targeted by higher energy laser pulses.
To address this issue, simultaneously to performing 16S rRNA PCR, we followed a strategy of systematic qPCR for lymph node specimens that targeted Bartonella spp., F. tularensis, T. whipplei, and Mycobacterium spp (3 ).
Given that CD147 and CD44v3-10 colocalise with MDR1-positive cells in CaP specimens, their targeting could potentially overcome drug resistance in the late stage of metastatic CaP.
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