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This sample volume surge was expected to be specimens of human origin (clinical specimens); the need for large-scale environmental sampling and testing had not been anticipated.
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Women can simply have one specimen taken, and reflex testing can be done on the specimens, avoiding the need for either a repeat visit or dual specimen collection.
HPV screening with HC II and HPV-typing analysis can be performed using residual specimens without the need to collect a second specimen from the patient; this collection system is thus of great advantage to both patients and clinicians.
To determine if pathologic findings in cone biopsy specimens correlate with residual invasive disease in radical hysterectomy specimens and the need for adjuvant chemo-radiation therapy.
Alternatively, when using CNS tests, it is possible to describe the failure curve of a specimen by obtaining the strength parameters c and ϕ with a single specimen, avoiding the need to prepare many specimens.
In addition, different samples are prepared and measured at varying times, but high tissue growth deviation between specimens and the need for monitoring tissue growth and scaffold degradation at different times requires large sample numbers for statistical analysis.
This approach would require examining a very large number of specimens, increase the need for hospitalization and would not be suitable for children in high burden settings.
Limitations in the availability of many clinical specimens drive the need for low DNA inputs into molecular assays.
The method allows numerous samples to be screened, because the microbial nucleic can be derived directly from human specimens, without the need for culture enrichment.
env class C was not amplified from any African specimens, obviating the need for the non-parametric test for panmixia for this gene.
In addition, the codetection of human RVA, a major cause of viral gastroenteritis in children, in both WUPyV-positive specimens underscores the need for further investigations to clarify the precise role of WUPyV in the pathogenesis of acute gastroenteritis.
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