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In this study, we observed abnormal Fhit protein expression in intramucosal carcinomas, with 38.2% of specimens demonstrating a decrease in, or absence of, Fhit protein staining.
In this study, we observed frequent abnormal Fhit protein expression in poorly-differentiated CRCs, with 77% of the specimens demonstrating a decrease in, or lack of, Fhit protein staining.
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The specimens demonstrated a typical electrolytic behaviour.
RESULTS: Under cyclic loading, the intact specimens demonstrated a significantly higher secant stiffness (74.8 ± 1.6 N/mm) than the repair groups (endosteal Pec Button [PB], 46.2 ± 6.4 N/mm; suture anchor [SA], 45.9 ± 8.7 N/mm; transosseous [TO], 44.2 ± 5.5 N/mm).
Other specimens demonstrate a slightly more advanced stage of development (Ba22, 30, 31; Figure 1D).
Histologic examination of the resection specimens demonstrated a pCR of the primary tumour in eight of 36 patients (22%).
RT PCR performed on three different glioblastoma surgical specimens demonstrated a heterogenous expression of the RAS components in human glioblastoma.
Early local recurrence specimens demonstrated a significantly increased infiltration of CD11b+ myeloid cells (P=0.0097) but decreased CD31-positive vessel density (P=0.0004) compared with their matched primary samples.
In vitro studies of COS7 cells overexpressing DHHC9 and colocalisation studies on FFPE specimens demonstrated a Golgi-restricted expression of DHHC9.
Measurement of radiolabelled antibody in the resected specimens demonstrated a 2.6-3.3 2.6-3.3crease in comparison with the surrounding normal tissue (P less than 0.01).
Thus, IVD cells from traumatic disc specimens demonstrated a significant increase of caspase-3/7 activity (RLU 8404.3 ± 2463.3) when compared to healthy control samples (RLU 706.1 ± 287; p < 0.001; Figure 2).
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