Under either Assumption 5 or when we postulate that the market model (mathcal {M}) is regular, we obtain the same conclusions as under Assumption 4 and thus they are not helpful when analyzing the valuation of nonreplicable contracts.
When we postulate conservative sorting in analogy to cyanobacteria, the cyanelle Sec translocases must reside in both thylakoid and inner envelope membranes.
It remains under-powered even when we postulate the moderate risk (power = 0.56 to 0.71 at a relative risk of 1.4 to 1.5 [ 6]).
Specifically, we postulate that arrivals and departures from stations can be separated into: (i) arrivals (and departures) due to consumers, and (ii) arrivals (and departures) due to the system operators for rebalancing the system.
Specifically, we postulate that 6bpΔ mutL cells pre-exist as rare variants in bacterial populations, rather than arising in response to environmental or metabolic challenge.
Specifically, we postulate that the UPR is normally able to maintain a protein folding equilibrium in the presence of aminoglycoside-induced mistranslation in SGCs.
Specifically, we postulate that an ancestral secreted form (α-subclass) of type-3 copper protein emerged early in the evolution of life.
Specifically, we postulate that the inefficient mitochondria damaged by chronic exposure to a HFD will be required to have increased O2 consumption in vivo to maintain adequate ATP levels in the liver tissue.
More specifically, we postulate here that P-DUDES proteins are involved in the EMT and may be involved in regulation of the interactions between the cell and the extracellular matrix.
Specifically, we postulated that the toxic effects of thymidine analogues might result in pyrimidine depletion, detectable as a reduction of systemic and/or fat uridine concentrations.
More specifically, we postulated that LPS acts at the luminal surface of the brain endothelial cell to induce release of soluble factors from its abluminal surface.
