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Rats with an isolated CA1 area showed normal acquisition of an associative hippocampal-dependent spatial recognition task.
counteracted the scopolamine [0.2 mg/kg, subcutaneously (s.c).]-induced non-spatial and spatial recognition memory deficits.
The disturbance in such elements, neurotransmitters, and enzymes may potentially impair the spatial recognition memory of experimental mice.
Moreover, the spatial recognition memory and locomotor activity were impaired mostly due to the inflammation response to TiO2 NPs.
For this purpose, the novel object task (NOT), a procedure assessing non-spatial recognition memory and the novel location task (NLT), a procedure evaluating spatial recognition memory were used.
The present study was designed to investigate the effects of ketamine on rats' non-spatial and spatial recognition memory.
The aim of the present study was to evaluate the effect of the monoaminergic stabilizer -OSU6162 on spatial recognition memory.
This left the picture well short of allowing for full 3D spatial recognition of a given field, though, as that horizontal plane would essentially go on forever, ignoring walls or other vertical surfaces.
Spatial recognition memory could be influenced by disturbance of the trace elements in mice' s CNS due to chronic exposure to TiO2 NPs as well.
These results suggested that the spatial recognition memory in mice should be impaired, due to long-term exposure of low-dose TiO2 NPs.
These findings suggested that an intrinsic apoptosis pathway in the mouse hippocampus was induced by TiO2 NP exposure, which resulted in impaired spatial recognition ability [62].
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