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Some obligates intracellular parasites, such as Encephalitozoon, Plasmodium, and Toxoplasma live in a parasitophorous vacuole in the host cell [ 94].
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As a result, some obligate and facultative grassland birds are projected to decline, while numerous species tolerant of or partially reliant on woody vegetation will increase.
As a result of genetic engineering, some obligate photoautotrophs, formerly unable to partake in a sweet diet, have been given a taste of heterotrophy through the introduction of hexose transporters [125].
Some obligate endosymbionts are obviously mutualistic and contribute to the fitness of their hosts, whereas other facultative endosymbionts are parasitic and often affect their hosts negatively [1] [3].
Such static conditions may apply to some obligate symbionts, which can indeed lack the ability to regulate genes involved in important metabolic processes.
Finally, identifying all components of any secretion system may be hampered when selection does not favor the clustering of all genes within one operon, an apparent characteristic of some obligate intracellular bacteria [150], [192].
Some obligate parasites, like Rafflesia, have lost photosynthetic capabilities and are fully heterotrophic.
Some obligate mutualisms are transmitted vertically; during reproduction, the host transmits symbiont offspring to its own offspring.
However, compared to some obligate symbionts of insects [ 24- 27], B. rhizoxinica has a 3-4 times larger genome (Table 1).
However, some obligate anaerobes can tolerate small amounts of oxygen thanks to the presence of some proteins involved in oxygen detoxification [ 10].
The bacterial density progressively increases along the small bowel from ∼10 in the jejunum to 10 colony-forming units per gram of luminal content at the ileal end, with a predominance of gram-negative aerobes and some obligate anaerobes (8).
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