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Notably, treatment of membranes with reagents, particularly those attacking sulfhydryl groups, was shown to enhance the sodium effect.
The primary efficacy endpoint in these studies was the mean change in serum sodium over the duration of treatment, as measured by the area under the serum sodium effect curve (AUC), corrected for baseline serum sodium.
The treatments were designed to isolate particular ion effects: NaCl, the salt of interest, CaCl2 to isolate the chloride effect, Na2SO4 to isolate the sodium effect, and the combination of NaCl + CaCl2 to represent a mixture of mono- and divalent cations that would be closer to a 'real' exposure.
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The sodium effects were evaluated by comparing the mechanical properties data in air and sodium.
Based on the mechanical performance of the steels in sodium, consideration of sodium effects in high temperature structural design of advanced fast reactors is discussed.
One final trick to outsmart sodium: Foods with potassium dampen sodium effects and even help relax your muscles.
It is thus not surprising that experimental and human studies assessing the effects of sodium bicarbonate effects in shock patients with severe lactic acidosis have not shown any improvement in cardiovascular function [ 82, 83, 87- 92].
Since the sodium void effect is strictly positive in this core and dominant in the transient, a power excursion is initiated by sodium boiling in the ULOF case.
In the study, the sodium void effect and the major feedback coefficients are analyzed.
Sodium environmental effects are key limiting factors in the high temperature structural design of advanced sodium-cooled reactors.
In patients with sodium channel effects (wide QRS complexes, shock), sodium bicarbonate can be used, and class IB anti-arrhythmics such as lignocaine can be used if sodium bicarbonate fails.
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