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However, because the primary tumor still involved the SMA, it was not resected.
Using SMA, it would be possible to determine the potential loading capacity of the anaerobic treatment systems, thus allowing appropriate organic loading rates to be applied (Javed and Tare 1999).
In a zebrafish model of SMA it was shown that the addition of purified snRNPs could rescue motoneuron impairment induced by the loss of SMN [5], [32].
For the two-pass SMA it was 40 bp for first-pass and 15 bp for second-pass.
While dopaminergic medication reinstated physiological γ-γ coupling from lPM to SMA, it did not normalize the task-related cortical coupling pattern.
To further understand the pathological events leading to SMA, it is critical to elucidate whether the SMN reduction in motor neurons is a primary defect in the disease or whether there are independent muscle defects as well.
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With the limitation of the conventional BRBs and the favorable feature of the SMAs, it has been questioned that if the SMA braces (SMABs) can be used as alternatives to the conventional BRBs to further reduce the residual inter-story drifts while limiting the maximum transient inter-story drifts in CBFs.
To avoid interfacial debonding between the SMA fiber and the epoxy matrix and to take full advantage of the properties of SMAs, it is necessary to investigate the internal stress distribution profiles.
For SMA 1, it was 6.00 %, whereas for SMA 2 it was slightly higher (6.12 %).
It should be pointed out however that there is no evidence of splicing defects in SMA and it is not known whether the reduction of snRNP levels reported here would indeed affect the functionality of the minor pre-mRNA splicing pathway.
Antegrade revascularization accomplishes less angulated, shorter bypass for CA and SMA, but it is unsuitable for high-risk cases because a supraceliac approach is necessary [15].
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