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ODCB exhibits the slowest membrane permeation of the analyte.
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This proof of concept study demonstrates that the controlled-release platform can be used to encapsulate a variety of compounds to slow membrane biofouling.
Often a secondary slow depolarizing phase was initiated by these spontaneous action potential events, resulting in slow membrane oscillations.
The interaction of gabaergic and non-NMDA glutamatergic systems spared by ketamine and xylazine generates slow membrane potential oscillations that drive cortical neurons periodically [30].
Even if the amplitude of membrane potential fluctuations remains constant, it is expected that the slow membrane potential changes induced by adaptation will result in the modification of one or the other power law parameters (slope or exponent).
Previous studies have shown that adult-born neurons in the subgranular zone of the dentate gyrus have slower membrane time constants, higher input resistance and more depolarized resting membrane potentials [26], [49].
In order to understand the cellular and network properties that modulate slow membrane potential fluctuations, it is often required to detect, separate and quantify the up and down states for further detailed data analysis.
The unanticipated observation that, even in the presence of high EPO, the lack of a large part of the receptor cytoplasmic domain results in the cell membrane accumulation of mutated EPOR, suggested that the truncated receptor has a slower membrane internalization/degradation and an increased half-life due to the absence of sequences required for receptor internalization and degradation.
This signal transduction pathway mediates slow membrane depolarization and vasoconstriction of canine mesenteric vein.
Young GCs have higher input resistances, slower membrane time constants than mature ones.
Furthermore, we identified a novel signal transduction mechanism governing the slow membrane depolarization, which involves enzymes of the PKC family.
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