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Exact(6)

Finally, the HIGH orders (and/or repetitions) (these moments describe the fine-image information) can be modified enough, since this modification will not be very visible.

For example, moments of LOW orders (and/or repetitions), these moments describe the coarse image information and LOW amplitudes should not be modified enough, since this modification will be visible to the final watermarked image.

Since this modification normally marks damaged DNA, which activates checkpoints, it is unclear why it persists on telomeres and whether its occurrence has functional consequences.

This finding is important since this modification of the senescent cell phenotype may alter the cells ability to withstand hyperthermia and other different types of physiologic stresses.

Since this modification labels enhancer elements [ 29], the association of SYT-SSX2 with these sites as well as Polycomb target sites suggests that SYT-SSX2 may effect transcription by modulating both enhancer and Polycomb function.

Since this modification is reported to be associated with enhancer activity (Heintzman et al. 2007), it is possible that the modification modulates the enhancer activity with the change of nucleosome positioning.

Similar(54)

Since then, this modification has been studied extensively due to its importance for the proper cellular activity of numerous proteins.

The analysis of auxin transport in Arabidopsis inflorescence stems suggested that this might indeed be the primary function of this modification since auxin transport was strongly impaired in d6pk loss-of-function mutants while PIN abundance at the plasma membrane was not altered.

Since this surface modification process produces exceptional corrosion resistance, it should be considered as a candidate for the replacement of chromate conversion coatings.

Since this histone modification is associated with enhanced gene activity, demethylation of H3 lysine 4 by JARID1C leads to transcriptional repression [6].

However since this histone modification might promote the recruitment of DSB repair proteins per se, it appears reasonable that critical compensatory mechanisms would be engaged to prevent the initiation of conflicting DNA damage/repair responses, i.e., in instances where no DSBs are actually induced.

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