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Alternatively, Kvanvig suggests, we may characterize Kp rigidly to say, 'there is an actual being x and actual time t such that it is known by x at t that p.' Since this expression designates rigidly (i.e., it makes reference to the actual world regardless of the modal context in which it appears), it may be substituted for p in the knowability principle.
Since this expression pattern was reminiscent of that of PROS, we analyzed the possible co-expression of pros and dap by double FISH.
Since this expression pattern is similar to what we have observed in human breast cancers [12], we chose the MMTV-PyMT mouse breast tumor model to test the functional role of MMP13 in breast cancer progression and metastasis.
Third, since this expression profiling experiment cannot distinguish the relative contribution of maternal versus zygotic mRNA to the steady state mRNA levels, it remains possible that maternal mRNAs that are not yet degraded by 24 hpf could mask changes of Spt5-dependent zygotic gene expression.
Since this expression pattern is similar to that described with PDA patients, 14 15 this mouse model represents a tractable experimental platform to interrogate the interaction of SPARC and nab-paclitaxel.
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Since then this expression will be 0 when.
Since ThiS fusion enhanced the expression of insulin chain A and B [ 13], we also fused the gene of MoaD to the gene encoding insulin chain A or B at their upstream and cloned into prokaryotic expression vector pET28a.
Since we will use this expression again later we will mark it with an asterisk.
Since this pleiotropic expression profile appears to be strictly partitioned between the two Lhx3/4 paralogs in M. occidentalis, this constitutes a clear example of regulatory sub-functionalization following gene duplication (Ohno, 1970; Hahn, 2009).
Since this latter expression profile evolves, after HAT release (passage 8), towards a return to a parental-like XCI pattern, these clones have probably locally and transiently reactivated both XP-linked loci together with the Hprt1 WT gene.
Since this limited expression of the proinflammatory key cytokine IL-1 β may barely explain the observed downstream effects as the neutrophil influx, complement components known to be involved in a variety of acute and chronic inflammatory diseases [ 21] were measured and found to be elevated.
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