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The stochastic simulations described here explored the roles of diffusion, PKA and PDE4s in generating spatial microdomains.
This so-called neutral scenario ([13], [16], more appositely a neutral-niche scenario) has no steady state outcomes in the analyses and simulations described here, because setting all αij = 0 (i≠j) allows indefinite expansion of S and hence also of N. A slightly less extreme neutral-niche community is modelled by setting all interspecific impacts to a common low value.
In the simulations described here: 1.
The simulations described here support and supplement such a model.
All estimators were implemented by the authors in SAS, and all simulations described here were also performed in SAS.
For the simulations described here, we defined action choices as the choice to send an SMS message addressing one of three possible reasons for medication non-adherence.
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If other PKA targets are essential for the microdomain, simulated block of PKA phosphorylation will not eliminate the microdomain because the simulation described here does not include these other PKA targets.
The simulation described here does not include coupling between the 16-bit alleles to form extended haplotypes that undergo independent assortment.
This approach is impractical in the empirical model described here as it would imply an infinite simulation of stochastically determined rotations.
Monte Carlo simulations (not described here) indicate that the NLS estimator attains this lower bound for s.
The surgical simulation tools described here operate on the surface definitions of the anatomical structures.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com