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3D genome simulation suggests a model of chromatin folding around chromosomal axes, where CTCF is involved in defining the interface between condensed and open compartments for structural regulation.
Theoretical simulation suggests a F/CF3 functionalization by formation of covalent bonds and also predicts a corresponding p-type doping and Dirac cone opening after the F/CF3 adsorption.
Analysis of particle morphologies in the simulation suggests a strong influence of temperature rise at the periphery of CS particles during deposition and dynamic recrystallization with the strong jetting of molten metal allowing for inter particle mixing and substrate adhesion.
MD simulation suggests a collapsed pore of F19P channels at the lower bilayer leaflet.
Notably, a recent study using computational simulation suggests a complex and reciprocal influence between recruitment and movement of Pol II on the ultimate transcriptional efficiency (Ehrensberger et al., 2013).
First, our model simulation suggests a small size of the domestication bottleneck but also a relatively short duration of the bottleneck, which implies a limited reduction in genetic diversity.
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The MD simulation suggests an approximately equal population of each conformer.
Using a set of real data, the simulation suggested a 3-period dMZD configuration for driver scheduling with preferable levels of tour length and imbalance statistics.
The D54 T82 interaction was present in 3 of 4 replicas, while the Y101 active rotamer was found in 2. Interestingly, except for one case, these two features were generally observed together in the same simulation, suggesting a correlation between them.
Nevertheless, the concept of software-hardware co-simulation suggests a new hybrid approach: the transceivers can be implemented in software, giving researchers absolute configuration control and flexibility, whereas the channel emulation can be run on hardware, thus accelerating channel emulation and the overall simulation process.
This number of alleles based on our simulations suggests a mutation rate of around 10−4 for our set of microsatellite loci (Fig. S5).
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