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In our simulation, each of these two channels is modeled as a linear time variant filter channel.
Note that to obtain the points in Figure3d for 0 velocity, we averaged multiple runs of the simulation, each of them for a different network topology chosen randomly from steady state topologies.
There are 54 (6×9) PC-IN modules in the simulation, each of which has one PC and two INs.
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At the beginning of the simulation, each molecule of the population is initialized with a random sequence.
For all simulations N = 800, α = w = 1 and the distributions are pooled from 10 simulations each of length 104 seconds.
The regressions are run using the data of obtained from all the 6400 simulations, each of which is treated as an observation.
For 150 rounds of simulations, each of these deletions occurred with a frequency of 146, 143 and 141 respectively.
Five repeat, unrestrained CG-MD simulations (each of 3 μs duration, with different initial velocities) were performed.
Our study has the fortune to be based on sampling of a long 3000 nanosecond simulation plus 32 wildtype binding simulations (each of 150 to 250 nanoseconds in length) and 16 simulations of the mutant form (each of 250 nanoseconds in length) totalling over 12 microseconds for the binding/inhibition part of our experiments alone.
Each simulation ran for 50,000 generations, with 10 runs of the simulation for each of the two games.
Even if the simulation of each of these factors may be easy to understand (e.g. the simulation of survival, which may be as simple as throwing a dice), the combination of various factors makes the models more complex.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com