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Galtung's 10 structuralist theory of violence would suggest that which patient characteristics are associated with silence depends on existing structures, rather than patients' assertiveness.
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It may die, or it may just suffer in silence, depending on how sensitive the species is.
Production of dsRNA in transitive silencing depends on cellular RNA-dependent RNA polymerase (RDR) activity, and the siRNAs generated are termed secondary siRNAs, while those that derive from the initial dsRNA trigger are termed primary siRNAs.
The success of RNAi gene silencing depends on the specificity of siRNAs for particular mRNA sequences.
This silencing depends on Piwi in the follicle cells surrounding the oocyte.
The biological significance of the guide/Rnt1p complex and its potential as an effective tool for gene silencing depends on the cleavage efficiency of this complex.
Several results indicate that RNA also influences chromatin structure as, first, chromatin-induced gene silencing depends on the RNAi machinery [14] [16] and, second, dosage compensation requires the contribution of specific non-coding RNAs [17], [18].
The mode of action in silencing depends on the recognition of target mRNAs by single stranded miRNAs that get incorporated into the RNA induced silencing complex (RISC).
Hypomethylated primordial germ cells appear to limit this risk by expressing a cohort of retrotransposon-suppressing genome-defence genes whose silencing depends on promoter DNA methylation.
Genetically, Piwi-mediated TE silencing depends on its nuclear localization, but not on its slicer activity (Klenov et al., 2011; Saito et al., 2010).
Thus, CMV promoter silencing depends on the presence of both Np95 and de novo Dnmts, but ensues well before de novo methylation of the promoter is detected.
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