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Furthermore, a significant ventricle enlargement (increase with 23.5%) could be measured in the T. gondii infected brains.
We examined the relationship between partial loss of the ependymal cell lining of the lateral ventricles to surface gliosis and ventricle expansion and found that significant ventricle surface gliosis can be linked directly to lateral ventricle expansion, independent of any injury or degenerative loss of brain tissue.
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Cardiovascular effects include a significant right ventricle upregulation of IL-1 β, TNF- α, IL-10, and CD14, and a left ventricle difference in TNF- α, and IL-10 in South versus North Mexico City residents, a key point in relation to the marked difference in pollutant profiles determined by the residence MCMA location [ 43 ].
Similarly, no significant correlation between ventricle size and the degree of apoptosis was found among individual meningitis groups (p > 0.05).
As indicated above, under normal conditions, we never observed periventricular gliosis in mice of any age, nor did we observe significant changes in ventricle volume.
This pairing was unique as the two subjects showed significant differences in ventricle volume, which gave us the rare opportunity to examine tissue quality along the entire surface of the lateral ventricles.
All groups showed continued enlargement of the ventricles during the therapeutic period, and all groups displayed significant increases in lateral ventricle size compared to the images before treatment began (all p < 0.05, t-tests; Tables 1 and 2; Figure 2).
The majority of proteins which had a significant difference in one ventricle compared to the other pathology also had a similar trend in the adjacent ventricle.
Consistent with previous research, there were significant correlations between third ventricle width and neuropsychological tests.
In our patient, we detected enlargement of the right ventricle and significant tricuspid regurgitation; however, we did not assess for right ventricular hypokinesis or pulmonary hypertension.
Identical immune challenge in late gestation resulted in significant expansion of 4th ventricle volume but did not disrupt sensorimotor gating.
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