Sentence examples for signature of function from inspiring English sources

As a refined signature of function, T Mtb-iNet showed a higher degree of positive correlation with all the four separated peripheral blood cell populations of PTB patients (i.e., neutrophils, monocytes, and CD4+ and CD8+ T cells) with the highest positive correlation with neutrophils (Table 2, Figure 8).

Similarly, all caspases have a distinct structural fold that is essential for their catalytic function and may be used as a structural signature of their function (Thornberry and Lazebnik, 1999).

Moreover, a recent genomic analysis from the NCCTG-N9831 trial demonstrated a strong association between increased relapse-free survival in adjuvant trastuzumab-treated patients and a signature of immune function genes, including IFN-γ and TNF-α [ 21].

The present study tested two key signatures of attentional function that have been widely implicated in autism: attentional disengagement and social orienting.

We conclude that relatively few AC genes have close paralogs in the Z. tritici genome, a signature of the potential function of many such genes that echoes our observations of expression and evolutionary constraint at these loci.

The signatures of these functions are: begin{aligned} textit{Map} ;text{ k1, v1)} rightarrow [text{ k2, v2)}],textit{{Reduce} ;(text{k2, [v2]}) rightarrow [text{ k3, v3)}] end{aligned}The map function is applied on every (k1, v1) pair and produces a list of intermediate (k2, v2) pairs.

Transitions between life stages of M. genalis display striking changes in the functional categories of expressed genes, and life stages show distinct signatures of molecular functions.

The activation of this gene set is consistent with the observed up-regulated levels of KRAS in tumors from both species and with the up-regulation of pathways such as 'PI3K/AKT' downstream of KRAS (see below). the 'PTEN loss of function' signature was identified in human breast tumors lacking PTEN protein expression [ 47].

Immunohistochemistry for the HIF1α-inducible proteins CA9 and Glut1, and for HIF1α itself, revealed moderate or strong expression of at least one of these markers in all but two of the tumours, verifying the well-described hypoxic signature associated with loss of function of pVHL.

This kind of setup is easily achieved with the use of code instrumentation libraries (e.g., Byteman, AspectJ), as long as we provide the signatures of the functions to be intercepted.

Further, it is safe to say that Pfam and PRINTS are probably comprehensive enough to report signatures of 'known' functions.