Sentence examples for signals in the absence of from inspiring English sources

Voss, M., Ingram, J. N., Haggard, P. & Wolpert, D. M. Sensorimotor attenuation by central motor command signals in the absence of movement.

Changes in the magnitude and fidelity of calcium signals in the absence of dFMRP likely contribute to defects in neuronal structure/function, leading to the hallmark learning and memory dysfunction of FXS.

With generalized theory it is shown, first, that previously introduced 'canonical' schemes, characterized by primary phases being multiples of π/4, afford maximal intensity of the desired absorptive signals in the absence of secondary phase shifts, and second, how primary phases can be adjusted to maximize the signal intensity provided that the secondary phase shifts are known.

Figures 17, 18, 19, and 20 show that the temporal processing score is close to zero for such signals in the absence of a target.

This study was designed to examine whether CD4/CD8 lineage choices are affected by the reduced TCR signals in the absence of Itk.

First, activation of Smad/TGFβ signals in the absence of LRP1 results in the disruption of elastic layers and a tortuous aorta [1], as well as increased expression of PDGF receptors (Figure 2 and 3) [20], [41], [41].

These data indicate that PTPRS may be involved in negative regulation of EGFR signaling in the absence of oncogenic activation of KRAS in CRC.

Changes in peripheral adiposity were associated with increases in basal hypothalamic leptin signaling in the absence of changes in leptin receptor expression in LV-IRAS rats.

However, factors such as the membrane environment, interaction with autoantibodies, and mechanical stretch are known to increase G protein signaling in the absence of AngII.

However, it is likely that Dkks are also able to modulate Wnt/β-catenin signaling in the absence of Krm, since Dkk1 can directly block the Lrp6-Wnt interaction (Semenov et al., 2001) (Figure 2b).

We conclude that (i) unopposed p75 signaling (in the absence of p55) reduces infarct size following acute ischemia/reperfusion injury in naive myocardium, whereas unopposed p55 signaling (in the absence of p75) has no effect; and (ii) the development of the infarct-sparing effects of the late phase of PC requires nonredundant signaling via both p55 and p75 receptors.