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Onshore three-dimensional (3D) normal fault propagation through basalt shows linkage of surface fractures that produce a fault with an irregular trace (Kaven and Martel 2007).
One of these subpopulations (X) shows linkage equilibrium and therefore evidence for frequent mating.
However, the present work is the only one that shows linkage of the disease gene region to noncompaction in a genome-wide analysis.
This figure only shows linkage groups with the most QTLs from both studies.
Figure 2 shows linkage maps drawn for all four parents for an example linkage group (LG 13).
Our results also indicated that none of the loci located on this segmental part shows linkage to the markers mapped to LG7.
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Three RFLP markers (RG493, RG901, and CD0344) and sdh1 genes showed linkage with the BPH resistance gene.
Linkage analysis of 158 F2 individuals showed linkage of the RM17713 and RM13 markers with the NUDA locus at distances of 7.7 and 7.2 cM, respectively (Figure2d).
Three pairs of -EcoR I-CTG/Mse I-CCT, -EcoR I-CTG/Mse I-CAT, and -EcoR I-AGT/Mse I-CCC showed linkage with aphis resistance.
None of these markers showed linkage with HJEB in the data analysed here.
Noteworthingly, a majority of pure BFIS families[14] [17] also showed linkage to the same ICCA genomic area.
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