In fact, at least one cold-triggered amylase activity was shown to be activated as highlighted by zymograms [ 38].
Depending on the TRPC subtype and the tissue involved, TRPC channels were shown to be activated by a variety of stimuli (including neurotransmitters) or to be constitutively active.
In squamous cell carcinoma of the pharynx and larynx, NOTCH1 downstream signaling has been shown to be activated.
The mTOR pathway was shown to be activated in most of the cell lines independent of AKT signaling.
Nuclear factor kappa B (NF-κB) has been shown to be activated in the intestine after traumatic brain injury (TBI), and results in gastrointestinal mucosal injury.
Following stimulation with IL-1β, SGCs have been shown to be activated, which in turn may influence the neuronal responses [36].
In rice, ten members of SnRK2 family have been shown to be activated by hyperosmotic stress through phosphorylation (Kobayashi et al. 2004).
Furthermore, the mTOR pathway is shown to be activated in tumor tissue from both humans and mice.
The p38 MAPK pathway has been shown to be activated by ROS [52].
For example, the insula has been shown to be activated if unfairness is being recognised [26].
The reservoir of dormant LTRCs was shown to be activated in response to inflammation, stress, or injury signals [28] [30].
