Dissolved Mn in the leachate is predominantly Mn II) (Additional file 1: Figure S4), and ~ 40% of the aqueous Fe is present as Fe(II) (data not shown), suggesting a substantial presence of complexed Fe(III) in solution.
While all MOKE-FORC results for continuous films exhibit only one distribution (not shown), suggesting a strong hard/soft coupling, antidot results present at least two positive FORC distributions, in addition to negative regions (Fig. 6).
Surprisingly, the cells were found to be intact (data not shown), suggesting a bacteriostatic rather than bacteriolytic effect.
The analysis per genotype showed dN/dS values >1 for genotypes A, G, Ia, and K (data not shown), suggesting a phenomenon of positive selection.
We also noted a ∼2 fold increase in the average size of the foci (data not shown), suggesting a specific recruitment to or local expansion of effector T cells in the SG foci.
Reducing levels of muscle Myosin heavy chain also had a non-maternal interaction with baz producing dead embryos with the scraps phenotype (data not shown), suggesting a possible late interaction between muscle tissue and the epidermis.
Notably, circulating IGF-I levels in the GHRH (lit/lit) mice are in the range seen in our Snord116del mice (data not shown) suggesting a common final mechanism for the growth retardation related to available circulating IGF-I.
PGRP-SB1 is strongly induced in the fly gut after oral infection with Gram-negative bacteria ([36] and data not shown), suggesting a role in the intestinal immune response.
Among surviving Ate1−/− CaggCreER mice, 10% (8 of 80) developed patches of red hair among their normally black hair, in contrast to identically TM-treated Ate1-containing mice (data not shown), suggesting a misregulation of melanocytes in Ate1-deficient mice.
By itself, fetuin-A stimulated the formation of LC3-containing punctate structures (data not shown), suggesting a possibility that fetuin-A inhibits HMGB1 release potentially by stimulating its autophagic degradation.
Interestingly, 20% of the analyzed E13.5 Dkk1+/− embryos displayed exencephaly with a collapsed ventricle due a neural tube closure failure (data not shown), suggesting a possible modulation of the Wnt/PCP pathway by Dkk1 in the midbrain.
