Sentence examples for shown as demonstrated from inspiring English sources

The CSPG-associated patches were digested following the treatment (data not shown) as demonstrated above for older animals.

Based on the cleavage pattern observed during prothrombin activation, Bi-VSP converts prothrombin into thrombin in a manner similar to the mammalian blood coagulation factor Xa [39] (data not shown), as demonstrated for snake venom prothrombin activators [17], [40].

Interestingly, the MFS-associated strains RM3206, RM4193, and RM4197 with distinct LOS locus classes A, B, and F, respectively, also failed to bind cholera toxin (data not shown), as demonstrated previously in a recent study showing a lack of cholera toxin binding by all MFS-associated strains [50].

Cells expressing TNF-α in RA synovial tissue were found in a similar location in serial sections (data not shown), as demonstrated previously [ 35].

Cell counting Kit-8 tests were used to ensure that the overexpression of brevican did not affect the proliferation of the transduced 293T, transduced U251, and transduced U87 cells (data not shown), as demonstrated previously by Hu et al. [ 15].

As expected, no activity was observed with dAMP (result not shown), as has been demonstrated previously for HIV-1 RT [16].

To this end, we first screened a panel of human NSCLC cell lines and found that none of them expressed IL-12RB2 mRNA (Fig. 2A) due to methylation of a CpG island in exon 1 (not shown), as previously demonstrated for different malignant cells [1], [9], [20].

Furthermore, we have observed uptake of L-[1-C]Gal L-[1-C]Gal L-[1-C]Galotato (data not shown), as already demonstrated in N. bynthamiana [ 19].

In comparison, the control in-vitro-cultured NRP-1 knockdown MSCs produced highly disorganized network assemblies (results not shown), as previously demonstrated (see Figures 7B and 7D).

As indicated above, genes for FtsX, FtsE, MinC, MinD, and MinE have been lost in the M. infernorum lineage, whereas the ftsZ gene apparently has experienced a period of accelerated evolution (data not shown) as previously demonstrated for the ftsZ genes of several Prosthecobacter species, which also belong to the Verrucomicrobia [ 40].

Our results suggest that in thyroid cells with BRAFV600E oncogenic activation the effect of sorafenib in apoptosis depends upon the balance in the levels of the anti-apoptotic proteins Mcl-1 and Bcl-2 and not in the levels of Bad and Bax (data not shown), as previously demonstrated in melanoma cells [ 11, 35, 36].