Multivariable logistic regression analysis identified three factors associated with fetal loss, namely maternal shock (odds ratio 6.85), maternal transfusion of blood products (odds ratio 7.24) and lower gestational age (odds ratio 1.2 per week less than 37).
Although not significant (p = 0.14) NS1 positivity was associated with a higher likelihood of developing shock (odds ratio = 1.98, 95% CI 0.83 to 4.8, likelihood ratio = 1.8).
Subjects were filmed in 3-dimensions, but analysis of shock transmission ratio (TR) was limited to the sagittal plane.
It is well known that the DSA process produces the power-law spectrum, (n( p sim p^{-Gamma }), with (Gamma =(r+2)/ r-1)), where r is the shock compression ratio.
From the two different sets of observations we derived an estimate for the shock compression ratio X, which turns out to be X = 2.4 ± 0.2 and X = 2.2 ± 0.1, respectively, for the first and second event.
In this study, CURSI scores were derived from confusion; urea > 7 mmol/L; respiratory rate ≥ 30/min; and a shock index ratio > 1.0, scoring one mark each.
Multivariate logistic regression identified that a PCT level ≥ 2.6 ng/ml was independently associated with the development of septic shock (odds ratio, 38.3; 95% confidence interval, 5.6 263.5; P < 0.001).
Clearly, in the French phase III trial [ 6], treatment with low-dose corticosteroid was also associated with reduced mortality in etomidate-free septic shock (odds ratio = 0.40, 95% CI = 0.19 0.82).
Low-dose corticosteroids (150 to 300 mg/day) did not show benefit on all-cause mortality in patients with severe sepsis and septic shock (risk ratio = 0.92 (95% CI = 0.79 to 1.06)).
When considering only patients who developed sepsis, carriers of this haplotype were more likely to develop septic shock (odds ratio, OR = 1.62, 95% CI 1.04, 2.50, P = 0.031) (Table 5).
