Sentence examples for shift the expression from inspiring English sources

In the general case, and in order to shift the expression pattern of the genes K symbols up and down we consider a pair of K symbol alphabets: Σ↑ and Σ↓.

Thus, it can be assumed that in the presence of IL-17 a subset of genes up-regulated by TNF-α may shift the expression profile towards "IL-1β like" properties [ 17].

In this case, and in order to shift the expression pattern of the genes K = 2 symbols up and down, we need to consider, for example, the K = 2 symbol alphabets Σ↑ = { V, W} and Σ↓ = { B, C}.

For example, one "brain-specific" miRNA, miR-124 [ 29], was described in a number of reports and was demonstrated to shift the expression profiles of HeLa cells towards the brain signature [ 30, 31].

We compute P(N e, p B ) ∪ N e, (p B )↑)) ∪ N e, (p B )↓)) as follows: where shift ∈ {1,..., K}, and K is the value used in e-CCC-Biclustering with scaled patterns to shift the expression patterns K symbols up and down.

In addition, possible subfunctionalization shifts the expression pattern trends of gene pairs.

Ploidy duplication neither shifted the expression profile of a weakly metastatic cell to that of a highly metastatic one, nor obviously changed the expression levels of signature genes for highly metastatic cells.

However, treatment of wild-type mice with WY14643 and OA shifted the expression of Cyp4F16 to more distal regions, whereas EPA and DHA treatment resulted in significant increased expression in more proximal segments.

Deletion of this element shifted the expression pattern from being neuron-specific to notochord-specific, which is the default pattern observed with the basal promoter plus intron-enhancer combination.

Indeed, shifting the expression of hsa-miR-155 and hsa-miR-503, two miRNAs inversely regulated by TNF-α, towards the level seen in myoblast differentiation, rescued the inhibitory effect of TNF-α on myotube formation.

It is striking that treatment with HA shifted the gene expression profiles in an anti-inflammatory and anticatabolic direction relative to the LPS-challenged control group.