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Kineococcus radiotolerans SRS30216 was isolated from HLW within a shielded cell work area at the SRS [2].
Kineococcus radiotolerans was isolated within a shielded cell work area containing highly radioactive nuclear waste at the U.S. Department of Energy's Savannah River Site in Aiken, SC, USA [1].
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Our analysis of ethanol effects on early development showed that yolk cell E-cadherin/microtubule cytoskeleton was disrupted; blastomere radial intercalation was reduced; and shield cell migration was disorganized.
By serving as principal cellular stores for Zn2+ and Cu+ they warrant the supply of these essential trace elements in growth and rescue processes and, by their high sequestration power, they shield cell components from deleterious bonding by highly reactive metals such as Cd2+ and overabundant Zn2+ and Cu+ [ 3, 4].
The KD DMPK mutant is unable to shield cells from oxidative stress (see Figure 1).
These microenvironments might effectively shield cells leading to a higher localized concentration of autocrine growth factors.
The tight functional interplay among DMPK, HK II and Src protects from oxidative insults, shielding cells from the risk of lethal PTP opening.
Indeed, TAp73β has a protective role in shielding cells against Th decrease, that is one of the main steps for the development of Parkinson Disease [ 116, 117].
Sart et al. (2010) indicated that porous microcarriers are more suitable for the propagation of mesenchymal stem cells than solid supports, and this was thought to be due to the protective effect of porous microcarriers shielding cells from shear.
Lung cancer cells can express apurinic/apyrimidinic endonuclease (APE1) [ 7], which is a vital protective gene that shields cells from oxidative stress- and hypoxia-induced apoptosis and necrosis, and this gene has the dual functions of DNA damage repair and oxidation-reduction, which maximally reduce cell damage by regulating the adaptability of organisms to oxidative stress and hypoxia [ 8].
In CD105-deficient cells, the inhibitory effects of TGF β on proliferation and migration were found to be enhanced (Li et al, 2000a, 2000b), thus implying that abundant expression of CD105 might shield cells from the inhibitory effects of TGF β.
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