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Type 2 diabetes mellitus and atherosclerosis are complex diseases sharing common antecedents like inflammation.
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The impressive correlation between cardiovascular disease and glucose metabolism alterations has raised the likelihood that atherosclerosis and type 2 diabetes may share common antecedents.
The impressive correlation between coronary artery disease and alterations in glucose metabolism has raised the hypotesis that atherosclerosis and diabetes may share common antecedents.
The impressive correlation between coronary artery disease (CAD) and alterations in glucose metabolism has raised the likelihood that atherosclerosis and type 2 diabetes may share common antecedents.
Although diabetes and atrial fibrillation undoubtedly share common antecedents such as hypertension, atherosclerosis, and obesity (9– 11), the confluence of these two conditions clearly warrants additional study.
Another example, is the link between pregnancy complications (such as gestational diabetes and intrauterine growth restriction) and cardiovascular disease (CVD), which may share common antecedents [ 25- 27].
The 'common soil' hypothesis suggests that diabetes and CVD share common antecedents [32].
It is known that cardiovascular risk factors precede the onset of DM2 [ 1], and it has been proposed that CVD and DM2 share common antecedents [ 2].
Our finding of the positive association between arsenic exposure and sICAM-1 supports that diabetes and CVD share common antecedents that may be affected by arsenic exposure.
It is likely that both IPV and non-partner violence share common antecedents, such as CSA in the case of women, or a history of antisocial personality and alcohol abuse among men, which may account for all or part of this association [ 23, 24] Women with children from previous relationships were at increased risk of experiencing IPV in most sites (12/14, with 2 significant ORs).
It has been postulated that preterm birth is linked to maternal CVD later in life through shared common biological antecedents or inflammatory processes leading to the formation of atherosclerosis.
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