Sentence examples for sgs containing from inspiring English sources

Experiments with polyA+ and β-actin mRNA revealed a decrease in SG occupancy in either the percentage of SGs containing mRNA or in the percentage of SG volume occupied by them (Table 1).

SGs containing both TDRD3 and TIA-1 were present in 100% of cells after 30 min. Similarly, cells pre-treated with the methylation inhibitor MTA all contained TDRD3- and TIA-1-positive SGs after 30 min of stress stimulus, although small differences were observed at shorter time points.

However, Pateamine A induced SGs contain the initiation factor eIF2 (absent in As-induced SGs) and Pateamine A itself.

In addition to a variety of specific RNA-binding proteins, SGs contain 40S but not 60S ribosomal subunits [2].

Approximately 79% of the analyzed SGs contained or interacted with mRNA granules, which occupied about 34% of their volume (Figure 9B and Table 1).

SGs contain the majority of polyadenylated mRNAs that are subject to stress-induced translational arrest but exclude mRNAs encoding stress-induced proteins.

The differentiation of SGs coincides with the expression of Kit, and only Kit− SGs contain SSCs [ 13– 15].

For example, a member of this family expressed in tabanid SGs contains a disintegrin (RGD) domain and functions as a platelet aggregation inhibitor [ 31, 32].

SGs contain a large number of RBPs that harbor low complexity sequence domains that nucleate through transient, low affinity interactions (Kato et al., 2012).

Only Kit-negative (Kit−) SGs contain the SSC population (the undifferentiated SGs), and Kit-positive (Kit+) SGs are the differentiating SGs, where Kit is important for migration, proliferation, and differentiation [ 13– 15].

SGs contained several cytokeratin transcripts corresponding to cells at differentiated or differentiating epidermal layers (KRT1, 2, and 10), whereas the KCs contained cytokeratins typical of cells that maintain their proliferative capacity (KRT5 and 14) or that are activated by wound healing, hyperproliferative skin diseases or in vitro culturing (KRT6, 16, and 17) [ 1, 20].