"severe division" is a correct and usable phrase in written English.
You can use it to describe a situation when two or more parties are divided in an extreme way, such as when there is intense hostility between them. For example, "The mayor's proposal has caused a severe division among city residents, with strong emotions on both sides."
In addition, the Δga5dh cells exhibited severe division defects, including abnormal septum position (Fig. 4A) and asymmetrical divisions (Fig. 4B).
On the other hand, there is a much more severe division block under these conditions in the absence of FipA.
Incidentally our results do not support the hypothesis of a specific role in ciliogenesis for Poc1 in Clytia, as demonstrated in zebrafish and human epithelial cells [7]; Poc1-Mo embryoshoweded severe division and morphology defects without apparently affecting ciliation of the ectoderm cells at the onset of gastrulation.
Whether he reads them carefully or read about them, he would have found in those authors strong evidence of national disunity, severe class division, decadence, alcoholism, and the need for transforming Russia by one means or another.
Discusses the severe divisions between men and women in Kombai society.
Mrs. Megawati was able to smooth the severe divisions in her cabinet last week and, finally, after a marathon cabinet session that ran until midnight last Friday, the government issued two new antiterrorism decrees that provide for sweeping powers of investigation and detention.
A significant skeletal component is usually present in severe Class II Division 1 malocclusions.
This study aimed to examine end-of-treatment outcomes of severe Class II Division I malocclusion patients treated with surgical or non-surgical approaches.
When deleted, ZapC causes a mild division-defective phenotype, but when overexpressed, ZapC causes a severe cell division defect leading to filamentous growth [14], [15].
In S. pneumoniae, deletion of divIVA results in a severe cell division defect, and two-hybrid experiments suggested that DivIVA interacts with a number of cell division proteins (Fadda et al, 2003).
Since non-polymerising FtsA mutants show a severe cell division defect, it was concluded that polymerisation of FtsA is indispensable for its function.
