Variability between different experimental sets is explained by the age of responding cells; we observed that later passaged NIH3T3 cells responded to CM and purified Wnt3a control protein more weakly than earlier passaged cells (compare Fig. 10b left vs. right).
The data available from these routinely collected data sets are explained in the footnote of table 1.
Our analysis also revealed that a very small amount of the variation in the symptom sets is explained by the exposure variables.
Our results also show that a very small amount of the variation in the symptom sets is explained by the exposure variables.
Once the dataset is reordered, we then test various a priori created gene sets to see how important each is vis-à-vis the reordering (construction of gene sets is explained in the next sub section).
The approach used to identify this parameter value set was explained in Sect.
As shown in Fig 2A, over half (53.2%) of the total variance in this data set was explained by four variables— tissue type, histology, diameter, and degree of dysplasia.
The off-set is explained by the model.
In this section the variation of IL value with size of data set is explained.
Initially, design procedures of the FEFSM including parts drawing, materials and conditions setting, and properties setting are explained.
