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In contrast, serum chloride concentrations remained normal after Hartmann's infusion [41].
The average serum chloride level was defined as the average of the 10 serum chloride levels in patients who did not develop AKI or developed AKI 72 hours after liver transplantation and the average of the preoperative serum chloride level and the serum chloride levels measured before the development of AKI in those who developed AKI within 72 hours.
Preoperative serum chloride levels were measured within 24 hours before the operation and postoperative serum chloride levels were routinely measured every 8 hours during the initial 72 hours after ICU admission (10 measurements).
In chronically hypercapnic patients, renal adaptation to hypercapnia relies on elevating plasma bicarbonate and reducing serum chloride [7].
The primary outcome was defined as the all-cause hospital death according to quintiles of serum chloride levels.
Objectives: The association between postoperative serum chloride levels and the incidence of immediate postoperative acute kidney injury in the ICU in liver transplant recipients were evaluated.
Serum chloride concentration was greater with − 11 mEq/100 g DM DCAD concentration, while serum cation anion difference increased linearly with increasing the DCAD concentration.
Serum chloride was high in calves fed + 110 DCAD diet, while serum (Na + K) − (Cl + S) increased linearly with increased DCAD level.
Rabbits exposed to high CO2 partial pressure (10%) for 52 56 h reabsorb bicarbonate through the Na+/HCO3 − co-transporter, resulting in increased serum bicarbonate and decreased serum chloride [23].
In this study, statistically significant correlation between serum chloride levels of the brain-dead donors and the onset of the function of kidney allografts in the recipients was not found.
Furthermore, serum chloride levels were not changed.
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