Sentence examples for sequestration of target from inspiring English sources

Taking advantage of the abundant ZrOH groups on the surface of GO, as well as the large surface of available GO for enhancing the sequestration of target organophosphorus-pollutants, the as-prepared UiO-67/GO composites exhibit enhancement of adsorption capacity toward glyphosate.

MicroRNA-mediated mRNA decay and sequestration of target mRNAs in P-bodies have been also proposed.

On the other hand, overloading lysosomes with cholesterol completely inhibited exocytosis, possibly due to decreased lysosome motility or abnormal sequestration of target and vesicular SNAREs (Fig. 2).

Base complementarity between miRNA and mRNA influences the final outcome of the repression (fig. 1 d), in which a perfect base pairing results in target degradation, whereas imperfect base pairing yields sequestration of a target (Bartel 2009).

This mechanism leads to degradation or sequestration of the target mRNAs; in both cases, no translation of the mRNA targets occurs (Waters and Storz, 2009).

Whereas in M-Phase, all γ-H2AX was located along the condensed chromosomes with no cytoplasmic signal, the other phosphorylated proteins remained in the cytoplasm and did not co-locate with the chromatin indicating that 32Hc-activated DNA-PK does not lead to sequestration of the target proteins at a specific locus (Fig. 3D).

This process requires the sequestration of the target protein within a double membrane, the autophagosome, the delivery of the autophagosome to the lysosome, fusion with the lysosome and the efficient degradation by lysosomal hydrolases.

In contrast, NK cell lymphocytosis and leukemia are associated with autoimmune syndromes, such as vasculitis and RA [ 47- 49], suggesting that peripheral-blood NK cell defects in systemic autoimmune syndromes may be due to the sequestration of NK cells in target tissues.

However, such conclusions must be tempered by the fact that reports of NK cell defects in patients with autoimmune diseases have been based almost exclusively on studies of peripheral blood samples, which would not distinguish between true deficits and sequestration of NK cells in target tissues.

Although these reports raise the possibility that autoimmunity may be associated with NK cell numeric or functional deficiencies, such conclusions must be tempered by the fact that these observations have been based primarily on studies of peripheral blood samples that cannot distinguish between true deficits and sequestration of NK cells in target tissues.

This assumption might be violated if there is differential sequestration of pulsed and unpulsed targets cells in other tissues (such as the lungs).